酒精相关的器官损伤机制——阐明酒精导致器官损伤的分子途径，器官可塑性和器官康复的机制。
Mechanisms of alcohol-related organ damage - research that elucidates molecular pathways through which alcohol produces organ damage, as well as mechanisms of organ plasticity and organ recovery.
因此通过理解突触的原始发育情形，我们也可能得以了解作为可塑性基础的分子机制。
So by understanding the initial development of synapses, we may also be getting at the molecular mechanisms underlying plasticity.
一氧化氮作为一种气体信使分子，参与神经系统的信息传递，对海马、小脑等神经元突触的可塑性和神经网络的构建产生重要影响。
As a gas message molecule, nitric oxide takes part in the information transfer in nervous system, which affects plasticity of synapse in hippocampus, and cerebellum and construction of neural network.
最近的证据显示基因和早期生活应激事件与抑郁症的发生存在密切关系，最终与神经可塑性的细胞分子机制汇聚。
Recent evidence shows that genes and early life stress are associated with depression, and the mechanisms are converged on those of neural plasticity.
本课程探讨神经元传导，可激发细胞膜上之离子通道，神经突触传导与神经突触可塑性的分子以及细胞机制。
Surveys the molecular and cellular mechanisms of neuronal communication. Coversion channels in excitable membrane, synaptic transmission, and synaptic plasticity.
从分子机制上阐明蛋白质-蛋白质相互作用是了解细胞动力学和可塑性的基础。
Molecular elucidation of protein-protein interaction is essential for understanding the cellular dynamics and plasticity.
提示神经细胞黏附分子和胶质细胞源性神经营养因子可能参与了脑缺血损伤后脑的可塑性变化过程且与功能恢复有关。
It is indicated that NCAM and GDNF may be involved in the changing process of cerebral plasticity after cerebral ischemia injury, also associated with the functional recovery.
链间的相互作用：聚合物的分子链可以自由地彼此滑动(热可塑性)或通过交键互相连接(热固性或弹性)。
Interchain interactions. The polymer chains can be free to slide past one another (thermo-plastic) or they can be connected to each other with crosslinks (thermoset or elastomer).
过去大量的研究集中在学习后海马神经元和突触的分子、细胞的可塑性变化。
Previous studies have intensively demonstrated the molecular and cellular modifications induced in hippocampal neurons and synapses after the implementation of task-learning.
过去大量的研究集中在学习后海马神经元和突触的分子、细胞的可塑性变化。
Previous studies have intensively demonstrated the molecular and cellular modifications induced in hippocampal neurons and synapses after the implementation of task-learning.
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