目的了解内毒素受体在肝星状细胞活化中的变化和作用。
Objective to investigate the role of endotoxin receptor expression in the activation of hepatic stellate cells.
目的 了解内毒素受体在肝星状细胞活化中的变化和作用。
To investigate the role of endotoxin receptor expression in the activation of hepatic stellate cells (HSCs).
目的探讨内毒素受体CD 14 C 15 9t基因多态性与烧伤后严重脓毒症易感性及患者预后的关系。
Objective to investigate the relation of a lipopolysaccharide receptor CD14C159T gene polymorphism to severe sepsis susceptibility and prognosis in patients with extensive burns.
PAF受体拮抗剂sri63—441对内毒素和PAF所致肺水肿和肺泡灌洗液细胞学变化有部分改善作用。
PAF receptor antagonist SRI63-441 showed partial protective effects on endotoxin and PAF induced lung edema and BALF cytological changes.
目的观察不同潮气量机械通气对大鼠肺组织内毒素CD14受体表达的影响。
Objective To investigate the effect of mechanical ventilation with different tidal volume on CD14 receptor in lung tissue.
结论：两种组胺受体拮抗剂均可缓解内毒素诱导的雏鸡肺损伤。
Conclusion: Histamine receptor antagonists can reduce the lung injury induced by endotoxin.
目的观察纳洛酮对内毒素刺激的大鼠离体支气管平滑肌细胞内皮素1和内皮素A受体基因表达的影响。
Objective To investigate the effect of naloxone on the expression of ET 1 and ET AR mRNA in cultured rat bronchial smooth muscle cells stimulated by LPS.
目的：探讨内毒素血症对大鼠脑内促甲状腺激素释放激素（TRH）与其受体含量及分布情况的作用。
Objective: To study the dose and location of thyrotropin releasing hormone(TRH) and its receptor in Wistar rats brain when early endotoxemia happened.
结论HIC呈内毒素无反应性，其表面内毒素相关受体tlr4、CD 14和MD 2不表达是其耐受内毒素作用的重要分子机制。
Conclusion There was no response of HICs to LPS stimulation, and no expression of TLR4, CD14 and MD-2 on HICs may be an important molecular mechanism by which they tolerated to lipopolysaccharide.
结论HIC呈内毒素无反应性，其表面内毒素相关受体tlr4、CD 14和MD 2不表达是其耐受内毒素作用的重要分子机制。
Conclusion There was no response of HICs to LPS stimulation, and no expression of TLR4, CD14 and MD-2 on HICs may be an important molecular mechanism by which they tolerated to lipopolysaccharide.
应用推荐