So, some scholar propose that CD163/HO-1 pathway is hopeful to play a critical role in atherosclerotic vulnerable plaque stabilization.
因此有学者提出，CD163/HO-1通路作为一个新的抗炎途径，有望在稳定动脉粥样硬化不稳定斑块中起着重要作用。
Atherosclerotic vulnerable plaque is considered as an important pathologic basis of acute coronary syndrome and sudden cardiac ischemic death.
动脉粥样硬化易损斑块是急性冠脉综合征和心脏缺血性猝死的重要病理基础。
The vulnerable atherosclerotic plaque of carotid artery directed to acute cerebrovascular incidence. So to accurately identify the vulnerable plaque can prevent stroke.
颈动脉易损斑块是脑血管急性事件的根源，因此，准确识别易损斑块可积极有效的干预脑卒中。
Acute coronary thrombus formation secondary to vulnerable atherosclerotic plaque is the main pathological basis of ACS.
冠状动脉不稳定斑块破裂继发急性血栓形成是ACS的主要病理基础。
ObjectiveTo investigate the roles of apoptosis and express ion of PCNA in human vulnerable atherosclerotic plaque.
目的探讨增殖细胞核抗原(pcna)的表达和细胞凋亡在人冠状动脉不稳定斑块中的作用。
ObjectiveTo investigate the roles of apoptosis and express ion of PCNA in human vulnerable atherosclerotic plaque.
目的探讨增殖细胞核抗原(pcna)的表达和细胞凋亡在人冠状动脉不稳定斑块中的作用。
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