方法应用原子力显微镜，对U251胶质瘤细胞系进行观测。
Methods T he cultured U251 glioma cell line was detected with atomic force microscope(AFM) .
目的：研究LRRC4基因在恶性脑胶质瘤细胞系中的表达情况。
Objective To examine the expression absence of LRRC4 gene in glioblastoma cell lines.
但是最近有研究表明，胶质瘤干细胞并不一定比传统胶质瘤细胞系抗拒。
However, some investigators demonstrated that glioma stem cells are radiosensitive with a defective DNA damage response compared with established glioma cell lines.
方法：利用人胶质瘤细胞系u251细胞构建裸鼠皮下异位胶质瘤移植模型。
Methods: Ectopicly subcutaneous glioma transplantation models in nude mice were established by injection of human glioma cell line U251 cells.
目的建立诺帝诱导人胶质瘤细胞系CHG - 5分化的蛋白质表达图谱并比较其表达差异。
Objective To establish the protein expression profile of human glioma cell line CHG-5 differentiation induced by Nordy and identify the differentially expressed proteins.
前言：目的观察正常星形胶质细胞和星形胶质瘤细胞系s WO - 38的细胞骨架的区别。
Objective: to observe the differences in the cytoskeleton of normal astrocytes and SWO-38 cell line.
并采用酶联免疫吸附试验，用原代培养的正常胶质细胞、胶质瘤细胞以及胶质瘤细胞系对这52例胶质瘤患者进行检测和筛选。
The 52 glioma patients were detected and filtered with primary cultured normal glial cells, glioma cells and glioma cell line by enzyme linked immunosorbent assay.
埃伯哈特和他的同事们研究了恶性胶质瘤的细胞系以发现γ-内分泌酶抑制剂的影响，这种药物可阻断促使肿瘤增长的Notch受体途径。
Eberhart and colleagues studied glioblastoma cell lines to investigate the effects of a gamma-secretase inhibitor, a medication that blocks the Notch receptor, on tumor growth.
然后进行U 251细胞系中肿瘤干细胞的凋亡机制的探讨，从而为脑胶质瘤的防治及肿瘤疫苗的研制提供新的策略和分子靶标。
Then, based on U251 cell lines tumor stem cell apoptosis mechanism will be discussed. Accordingly provide new strategies for the therapy in glioma.
然后进行U 251细胞系中肿瘤干细胞的凋亡机制的探讨，从而为脑胶质瘤的防治及肿瘤疫苗的研制提供新的策略和分子靶标。
Then, based on U251 cell lines tumor stem cell apoptosis mechanism will be discussed. Accordingly provide new strategies for the therapy in glioma.
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