结论补体及炎症反应参与脑组织缺血再灌注损伤。
Conclusions Complement and inflammatory reaction participated in cerebral ischemia-reperfusion injury.
目的：观察血塞通改善脑组织缺血、缺氧的疗效。
Objective: It is to evaluate the therapeutic effect of panax Notoginseng saponins on improving ischemic and anoxia of the brain tissue.
结论脑泰通颗粒可改善大鼠脑组织缺血缺氧状态，具有保护脑组织作用。
Conclusion Naotaitong granule may improve anoxemia in rats brain and protect brain tissue.
目的：探讨脑醒冲剂对大鼠脑组织缺血再灌注的保护作用及其可能的机制。
Objective: To explore the protective effect of Naox - ing Instant Granules (NIG) on reperfusion of cerebral ischemia (RCI) in rats.
目的：研究浅刺针法对脑梗塞大鼠脑组织缺血半暗带神经细胞的保护作用及其机制。
Objective: to study protective mechanisms of superficial needling to ischemic penumbra neuron in cerebral infarction rats.
小纤维蛋白血栓可在脑、心脏、肺、肾以及其它器官的小动脉中形成，引起组织缺血。
Small fibrin thrombi can form in small arteries of brain, heart, lungs, kidneys, and other organs to produce ischemic tissue damage.
结论局部振动所致的组织中血管内皮活性物质的变化，可能与局部组织缺血、功能紊乱有关。
Conclusions Changes of vascular endothelial substance of tissues induced by local vibration may be related to tissue ischaemia and dysfunction.
微栓塞能够引起组织缺血，红细胞片断引发溶血性贫血，或者血小板和凝血因子消耗导致出血。
Microscopic thrombi can produce tissue ischemia, red cell fragmentation leading to a hemolytic anemia, or hemorrhage due to consumption of platelets and clotting factors.
结论早期使用高氧液可降低血液粘度，改善微循环，有助于改善脑组织缺血缺氧，减轻脑水肿。
Conclusion Early application of hyperoxia liquid can improve microcirculation, and decrease brain anoxia and brain edema.
应用钙离子拮抗剂在组织缺血再灌注损伤过程中可以抑制钙离子内流，降低组织细胞的破坏程度。
The application of calcium antagonists during ischemia reperfusion injury can inhibit the inward flow of calcium ions and decrease the severity of histiocytic destruction.
结论：二氢槲皮素能有效抑制ISO所致心肌组织缺血，保护心肌细胞免受损伤，从而起到治疗冠心病的作用。
Conclusion: Dihydroquercetin had an effective role to treat coronary heart disease by inhibiting myocardial tissue ischemia induced by ISO and protecting myocardial cells from injury.
结果：所有患者病变位于原翼状胬肉切除部位，且局部表现为组织缺血、变薄，部分患者病变区域可透见深层色素膜。
RESULTS: the ischemic, thinning could be found at the tissue where the pterygium was excised, and the deep pigment tissue appeared in some patients.
结果迟发性颅内血肿、弥漫性脑肿胀、侧裂区脑挫裂伤、脑组织缺血、缺氧等是重型颅脑损伤术中急性脑膨出的主要原因。
Results The main cause of acute encephalocele were delayed intracranial hematomas, acute diffuse brain swelling, cerebral contusion and laceration in Sylvain tissue and ischemic anoxia.
发病机制的研究涉及生物化学、分子生物学、血液异常的等方面，但其中心环节是高血糖和其引起组织缺血缺氧发生的一系列改变。
The research of the mechanism involves in biochemistry, molecular biology and blood dyscrasia, but it's center still is high blood sugar which causes a series changes of ischemia and hypoxia.
皮肤最里层的部分叫皮下组织，包含狭长的脂肪层——作为缺血时的储备。
The skin's base layer is the subcutis, which includes a seam of fat laid down as a fuel reserve in case of food shortage.
在这，研究人员发现缺血的出现，与心外膜脂肪组织量关系密切程度要高于冠状动脉钙评分。
Here, the researchers found that the presence of ischemia correlated more closely with epicardial adipose tissue volume than with the coronary calcium score.
目的检验口服叶酸能否改善高脂血症(HC)对缺血组织血管新生的抑制。
Objective to examine whether oral folate supplementation would rescue hypercholesterolemia (HC) -related impairment of angiogenesis in response to tissue ischemia.
目的：已有事实表明高血糖症可通过减弱血管扩张，提高组织酸度，释放刺激性产物导致脊髓的缺血损伤。
Hyperglycemia has been shown to potentiate ischemic injury of the spinal cord by quenching vasodilators and potentiating tissue acidosis and free radical production.
结果丹黄通络胶囊能提高急性不完全性脑缺血模型的大鼠脑组织中的SOD和伊文思兰的含量，降低LPO含量。
Results the Danhuang capsule can increase SOD AND Evans blue content of brain tissue from rat model of acute incomplete cerebral ischemia, at the same time, decrease the LPO content.
缺血性坏死：死亡的组织中，由于亏损的含氧血液，以一个地区的机构。
Ischemia necrosis: death of tissue due to loss of oxygenated blood to an area of the body.
因此溶栓同时联合抑制炎症反应，可能对减轻缺血脑组织的损伤有益。
Therefore thrombolytic therapy combined with preventing inflammatory response might contribute to relieving cerebral tissue damage after ischemia.
结果：氟桂嗪治疗组在再灌注各个时间点脑组织损伤程度均明显轻于脑缺血组。
RESULTS: in flunarizine therapy group, the degree of injury of brain tissue was significantly lighter than that at each time in cerebral ischemia group.
背景：幼鼠脑缺氧缺血后，脑组织水肿加重，脑组织中一氧化氮及丙二醛水平增高。
BACKGROUND: After cerebral tissue ischemia and anoxia in young rats, the cerebral edema gets serious, and the levels of nitric oxide (no) and malondialdehyde (MDA) decrease.
目的观察中药复方温心汤对家兔心肌缺血再灌注损伤心肌细胞凋亡以及病理组织学改变的影响。
Objective To observe mainly the effect of the Chinese traditional medicine (compound Wenxin soup) on myocyte apoptosis and path-histology change in rabbits ischemia referpusion injury.
结论血栓通能减轻脑组织的缺血再灌注损伤，改善神经功能缺失，其作用机制可能与增加HSP70表达有关。
Conclusion Xueshuantong can protect brain tissue from cerebral ischemia reperfusion injury and improve the nerve function. The mechanism may be related with the increasing of HSP70 expression.
结论血栓通能减轻脑组织的缺血再灌注损伤，改善神经功能缺失，其作用机制可能与增加HSP70表达有关。
Conclusion Xueshuantong can protect brain tissue from cerebral ischemia reperfusion injury and improve the nerve function. The mechanism may be related with the increasing of HSP70 expression.
应用推荐