内皮细胞功能障碍可造成心脏血管变窄变硬。
Dysfunction in endothelial cells contributes to the narrowing and hardening of heart arteries.
研究背景：内皮细胞功能障碍继发于高血压相关的血管损害。
Background - Endothelial dysfunction predisposes to vascular injury in association with hypertension.
内皮细胞功能障碍是在导致被称为的动脉硬化的血管闭塞的过程中起着关键作用。
Endothelial cell dysfunction plays a key role in the development of clogged arteries, a process called atherosclerosis.
因此，登革病毒所致的血管内皮细胞功能障碍，可能是DHFDSS重要的发病机制。
Therefore, functional impairment of vascular endothelial cells induced by dengue virus might be an important aspect in the pathogenesis of DHF/DSS.
结论OSAHS患者无论是否合并心脑血管疾病，均存在明显的血管内皮细胞功能障碍。
Conclusions OSAHS patients, no matter whether complicated by cardiovascular diseases or not, have significant vascular endothelial cell dysfunction.
微量蛋白尿是肾脏疾病及糖尿病并发症的一个危险因素，同时也被认为能反映内皮细胞功能障碍，这会致随后的血压升高。
Microalbuminuria is a risk factor for kidney disease and diabetic complications and is also believed to reflect endothelial cell dysfunction, which can later lead to high blood pressure.
目的：观察活血化瘀中药对多脏器功能障碍综合征(MODS)患者血管内皮细胞的保护作用。
Objective: To study the protective effect of activating microcirculation and removing stasis medical herbs on the vascular endothelium of multiple organ dysfunction syndrome (MODS) patients.
结论：OFR通过增加ADMA导致内皮功能紊乱，卡托普利则能通过减少ADMA减轻OFR诱导的内皮细胞代谢功能障碍。
CONCLUSIONS: OFR induces endothelial dysfunction through increasing ADMA concentration, while captopril relieves endothelial dysfunction induced by ox-LDL through decreasing ADMA concentration.
目的：观察多器官功能障碍综合征(MODS)大鼠肺组织中循环内皮细胞(CEC)以及细胞间黏附分子(ICAM - 1)和血管细胞黏附分子(VCAM - 1)的表达，并探讨它们之间的相互关系。
Objective: to observe the MODS rat lung tissue's CEC, as well as adhesion molecules ICAM-1 and VCAM-1 expression changes, and to investigate the relationship between them.
目的：观察多器官功能障碍综合征(MODS)大鼠肺组织中循环内皮细胞(CEC)以及细胞间黏附分子(ICAM - 1)和血管细胞黏附分子(VCAM - 1)的表达，并探讨它们之间的相互关系。
Objective: to observe the MODS rat lung tissue's CEC, as well as adhesion molecules ICAM-1 and VCAM-1 expression changes, and to investigate the relationship between them.
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